OxyContin 60 mg prolonged release tablets

OxyContin 60 mg prolonged release tablets

OxyContin 60 mg prolonged release tablets
(10 mg, 15 mg, 20 mg, 30 mg, 40 mg, 60 mg, 80 mg)

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Oxycodone hydrochloride
Chemical name: 4,5-epoxy-14-hydroxy-3-methoxy-17-methylmorphinan-6-one
hydrochloride
CAS No.: 124-90-3

DESCRIPTION
Oxycodone hydrochloride is a white, crystalline, odourless powder readily soluble in water,
sparingly soluble in ethanol and nearly insoluble in ether.
OxyContin tablets are modified release tablets designed to provide delivery of oxycodone
over 12 hours.
OxyContin 10 mg, 15 mg, 20 mg, 30 mg, 40 mg, 60 mg1
and 80 mg tablets have been
reformulated, and comprise a matrix formulation with a hydrogelling property (i.e. particles or
whole tablets become highly viscous (gel-like) in water), intended to be crush-deterrent and to
reduce the rapid release of oxycodone upon accidental or intentional misuse. The tablets have
been heat-treated to increase the mechanical strength of the tablet.
The physical properties of the reformulated OxyContin tablets were examined following an
extensive battery of physical manipulations. Beyond demonstrating that the reformulated
OxyContin tablets are harder to crush than another controlled release oxycodone formulation,
testing over the range of the reformulated OxyContin tablets fragment sizes showed that some
of the controlled.

OxyContin 60 mg prolonged release tablets is a full opioid agonist with no antagonist properties whose principal therapeutic
action is analgesia. It has an affinity for kappa, mu and delta opiate receptors in the brain and
spinal cord. Oxycodone is similar to morphine in its action. Other pharmacological actions of
oxycodone are in the central nervous system (CNS: respiratory depression, antitussive,
anxiolytic, sedative and miosis), smooth muscle (constipation, reduction in gastric, biliary and
pancreatic secretions, spasm of sphincter of Oddi and transient elevations in serum amylase)
and cardiovascular system (release of histamine and/or peripheral vasodilatation, possibly
causing pruritus, flushing, red eyes, sweating and/or orthostatic hypotension).
Opioids may influence the hypothalamic-pituitary-adrenal or –gonadal axes. Some changes
that can be seen include an increase in serum prolactin and decreases in plasma cortisol and
testosterone. Clinical symptoms may manifest from these hormonal changes.

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